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CC74A Rabbit Polyclonal Antibody, 100ul Endonucleases & Exonucleases The alternative splicing demonstrated in

CC74A Rabbit Polyclonal Antibody, 100ul Endonucleases & Exonucleases The alternative splicing demonstrated in

Name: CC74A Rabbit Polyclonal Antibody, 100ul Endonucleases & Exonucleases The alternative splicing demonstrated in
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CC74A Rabbit Polyclonal Antibody, 100ul Endonucleases & Exonucleases The alternative splicing demonstrated in

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Description

The alternative splicing demonstrated in Shank genes has been suggested as a mechanism for regulating the molecular structure of Shank and the spectrum of Shank-interacting proteins in the postsynaptic densities of the adult and developing brain

Defects in this gene are a cause of benign familial neonatal convulsions type 1 (BFNC)

Plexins are transmembrane receptors for semaphorins

Ig-alpha and Ig-beta

CC74A Rabbit Polyclonal Antibody, 100ul Endonucleases & Exonucleases The alternative splicing demonstrated in

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